deferoxamine dfo Search Results


deferoxamine (dfo  (AbMole Bioscience)
90
AbMole Bioscience deferoxamine (dfo
Deferoxamine (Dfo, supplied by AbMole Bioscience, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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deferoxamine (dfo)  (ApexBio)
90
ApexBio deferoxamine (dfo)
Deferoxamine (Dfo), supplied by ApexBio, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/deferoxamine+dfo/deferoxamine+mesylate++dfo/pm39706856-223-17-20
Average 90 stars, based on 1 article reviews
deferoxamine (dfo) - by Bioz Stars, 2026-10
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deferoxamine dfo m5129  (AbMole Bioscience)
90
AbMole Bioscience deferoxamine dfo m5129
Curcumin inhibits FTC tumorigenesis. (a) Nthy-ori-3-1 and FTC cells were treated with the indicated concentrations of curcumin for 24 h, and curcumin inhibited cell viability. Cell viability was measured using CCK-8 assays. (b–d) Curcumin suppressed the growth of FTC cells. FTC cells were treated with 0, 10, or 20 μ M curcumin and subjected to colony formation, cell migration, and invasion assays. Scale bars: 100 μ m. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the 0 μ M curcumin group. (e, f) FTC cells were either preincubated with various inhibitors, namely, ferrostatin-1 (Fer-1, 10 μ M), <t>deferoxamine</t> (DFO, 10 μ M), chloroquine (CQ, 5 μ M), autophagy inhibitor (3MA, 1 mM), and Z-VAD-FMK (5 μ M), for 2 h and then treated with 10 or 20 μ M curcumin (Cur) for 24 h or treated with curcumin alone. Cell viability was assessed using CCK-8 assays. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the group receiving curcumin treatment alone. The data are presented as the mean ± standard error of the mean (SEM), n = 3. One-way ANOVA (b–d) and t test (e, f) were used to determine statistical significance.
Deferoxamine Dfo M5129, supplied by AbMole Bioscience, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/deferoxamine+dfo/deferoxamine+dfo+m5129/pmc09867595-54-6-13
Average 90 stars, based on 1 article reviews
deferoxamine dfo m5129 - by Bioz Stars, 2026-10
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deferoxamine (dfo) mesylate  (FUJIFILM)
90
FUJIFILM deferoxamine (dfo) mesylate
Curcumin inhibits FTC tumorigenesis. (a) Nthy-ori-3-1 and FTC cells were treated with the indicated concentrations of curcumin for 24 h, and curcumin inhibited cell viability. Cell viability was measured using CCK-8 assays. (b–d) Curcumin suppressed the growth of FTC cells. FTC cells were treated with 0, 10, or 20 μ M curcumin and subjected to colony formation, cell migration, and invasion assays. Scale bars: 100 μ m. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the 0 μ M curcumin group. (e, f) FTC cells were either preincubated with various inhibitors, namely, ferrostatin-1 (Fer-1, 10 μ M), <t>deferoxamine</t> (DFO, 10 μ M), chloroquine (CQ, 5 μ M), autophagy inhibitor (3MA, 1 mM), and Z-VAD-FMK (5 μ M), for 2 h and then treated with 10 or 20 μ M curcumin (Cur) for 24 h or treated with curcumin alone. Cell viability was assessed using CCK-8 assays. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the group receiving curcumin treatment alone. The data are presented as the mean ± standard error of the mean (SEM), n = 3. One-way ANOVA (b–d) and t test (e, f) were used to determine statistical significance.
Deferoxamine (Dfo) Mesylate, supplied by FUJIFILM, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 90 stars, based on 1 article reviews
deferoxamine (dfo) mesylate - by Bioz Stars, 2026-10
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dfo-toc (deferoxamine-c6-dphe-cys-tyr-dtrp-lys-thr-cys-thr-ol, peptide purity = 92.9%, molecular weight = 1901.3 g/mol)  (CPC Scientific)
90
CPC Scientific dfo-toc (deferoxamine-c6-dphe-cys-tyr-dtrp-lys-thr-cys-thr-ol, peptide purity = 92.9%, molecular weight = 1901.3 g/mol)
Curcumin inhibits FTC tumorigenesis. (a) Nthy-ori-3-1 and FTC cells were treated with the indicated concentrations of curcumin for 24 h, and curcumin inhibited cell viability. Cell viability was measured using CCK-8 assays. (b–d) Curcumin suppressed the growth of FTC cells. FTC cells were treated with 0, 10, or 20 μ M curcumin and subjected to colony formation, cell migration, and invasion assays. Scale bars: 100 μ m. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the 0 μ M curcumin group. (e, f) FTC cells were either preincubated with various inhibitors, namely, ferrostatin-1 (Fer-1, 10 μ M), <t>deferoxamine</t> (DFO, 10 μ M), chloroquine (CQ, 5 μ M), autophagy inhibitor (3MA, 1 mM), and Z-VAD-FMK (5 μ M), for 2 h and then treated with 10 or 20 μ M curcumin (Cur) for 24 h or treated with curcumin alone. Cell viability was assessed using CCK-8 assays. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the group receiving curcumin treatment alone. The data are presented as the mean ± standard error of the mean (SEM), n = 3. One-way ANOVA (b–d) and t test (e, f) were used to determine statistical significance.
Dfo Toc (Deferoxamine C6 Dphe Cys Tyr Dtrp Lys Thr Cys Thr Ol, Peptide Purity = 92.9%, Molecular Weight = 1901.3 G/Mol), supplied by CPC Scientific, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 90 stars, based on 1 article reviews
dfo-toc (deferoxamine-c6-dphe-cys-tyr-dtrp-lys-thr-cys-thr-ol, peptide purity = 92.9%, molecular weight = 1901.3 g/mol) - by Bioz Stars, 2026-10
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deferoxamine dfo t1637  (Topscience Co Ltd)
90
Topscience Co Ltd deferoxamine dfo t1637
Curcumin inhibits FTC tumorigenesis. (a) Nthy-ori-3-1 and FTC cells were treated with the indicated concentrations of curcumin for 24 h, and curcumin inhibited cell viability. Cell viability was measured using CCK-8 assays. (b–d) Curcumin suppressed the growth of FTC cells. FTC cells were treated with 0, 10, or 20 μ M curcumin and subjected to colony formation, cell migration, and invasion assays. Scale bars: 100 μ m. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the 0 μ M curcumin group. (e, f) FTC cells were either preincubated with various inhibitors, namely, ferrostatin-1 (Fer-1, 10 μ M), <t>deferoxamine</t> (DFO, 10 μ M), chloroquine (CQ, 5 μ M), autophagy inhibitor (3MA, 1 mM), and Z-VAD-FMK (5 μ M), for 2 h and then treated with 10 or 20 μ M curcumin (Cur) for 24 h or treated with curcumin alone. Cell viability was assessed using CCK-8 assays. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the group receiving curcumin treatment alone. The data are presented as the mean ± standard error of the mean (SEM), n = 3. One-way ANOVA (b–d) and t test (e, f) were used to determine statistical significance.
Deferoxamine Dfo T1637, supplied by Topscience Co Ltd, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/deferoxamine+dfo/deferoxamine+dfo++t1637/pmc08811259-72-11-17
Average 90 stars, based on 1 article reviews
deferoxamine dfo t1637 - by Bioz Stars, 2026-10
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deferoxamine dfo 150  (Microm International GmbH)
90
Microm International GmbH deferoxamine dfo 150
Curcumin inhibits FTC tumorigenesis. (a) Nthy-ori-3-1 and FTC cells were treated with the indicated concentrations of curcumin for 24 h, and curcumin inhibited cell viability. Cell viability was measured using CCK-8 assays. (b–d) Curcumin suppressed the growth of FTC cells. FTC cells were treated with 0, 10, or 20 μ M curcumin and subjected to colony formation, cell migration, and invasion assays. Scale bars: 100 μ m. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the 0 μ M curcumin group. (e, f) FTC cells were either preincubated with various inhibitors, namely, ferrostatin-1 (Fer-1, 10 μ M), <t>deferoxamine</t> (DFO, 10 μ M), chloroquine (CQ, 5 μ M), autophagy inhibitor (3MA, 1 mM), and Z-VAD-FMK (5 μ M), for 2 h and then treated with 10 or 20 μ M curcumin (Cur) for 24 h or treated with curcumin alone. Cell viability was assessed using CCK-8 assays. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the group receiving curcumin treatment alone. The data are presented as the mean ± standard error of the mean (SEM), n = 3. One-way ANOVA (b–d) and t test (e, f) were used to determine statistical significance.
Deferoxamine Dfo 150, supplied by Microm International GmbH, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/deferoxamine+dfo/deferoxamine+dfo+150/10__1002_slash_jbmr__5650181305-721-0-7
Average 90 stars, based on 1 article reviews
deferoxamine dfo 150 - by Bioz Stars, 2026-10
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deferoxamine (dfo) bfc (p-ncs-bz-dfo)  (CheMatech Inc)
90
CheMatech Inc deferoxamine (dfo) bfc (p-ncs-bz-dfo)
Curcumin inhibits FTC tumorigenesis. (a) Nthy-ori-3-1 and FTC cells were treated with the indicated concentrations of curcumin for 24 h, and curcumin inhibited cell viability. Cell viability was measured using CCK-8 assays. (b–d) Curcumin suppressed the growth of FTC cells. FTC cells were treated with 0, 10, or 20 μ M curcumin and subjected to colony formation, cell migration, and invasion assays. Scale bars: 100 μ m. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the 0 μ M curcumin group. (e, f) FTC cells were either preincubated with various inhibitors, namely, ferrostatin-1 (Fer-1, 10 μ M), <t>deferoxamine</t> (DFO, 10 μ M), chloroquine (CQ, 5 μ M), autophagy inhibitor (3MA, 1 mM), and Z-VAD-FMK (5 μ M), for 2 h and then treated with 10 or 20 μ M curcumin (Cur) for 24 h or treated with curcumin alone. Cell viability was assessed using CCK-8 assays. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the group receiving curcumin treatment alone. The data are presented as the mean ± standard error of the mean (SEM), n = 3. One-way ANOVA (b–d) and t test (e, f) were used to determine statistical significance.
Deferoxamine (Dfo) Bfc (P Ncs Bz Dfo), supplied by CheMatech Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/deferoxamine+dfo/para++isothiocyanatobenzyl+deferoxamine+p++ncs+bz+dfo/pmc08446777-42-1-8
Average 90 stars, based on 1 article reviews
deferoxamine (dfo) bfc (p-ncs-bz-dfo) - by Bioz Stars, 2026-10
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dfo reference standard usp deferoxamine mesylate reference standard lot h  (Pharmacopeia Inc)
90
Pharmacopeia Inc dfo reference standard usp deferoxamine mesylate reference standard lot h
Curcumin inhibits FTC tumorigenesis. (a) Nthy-ori-3-1 and FTC cells were treated with the indicated concentrations of curcumin for 24 h, and curcumin inhibited cell viability. Cell viability was measured using CCK-8 assays. (b–d) Curcumin suppressed the growth of FTC cells. FTC cells were treated with 0, 10, or 20 μ M curcumin and subjected to colony formation, cell migration, and invasion assays. Scale bars: 100 μ m. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the 0 μ M curcumin group. (e, f) FTC cells were either preincubated with various inhibitors, namely, ferrostatin-1 (Fer-1, 10 μ M), <t>deferoxamine</t> (DFO, 10 μ M), chloroquine (CQ, 5 μ M), autophagy inhibitor (3MA, 1 mM), and Z-VAD-FMK (5 μ M), for 2 h and then treated with 10 or 20 μ M curcumin (Cur) for 24 h or treated with curcumin alone. Cell viability was assessed using CCK-8 assays. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the group receiving curcumin treatment alone. The data are presented as the mean ± standard error of the mean (SEM), n = 3. One-way ANOVA (b–d) and t test (e, f) were used to determine statistical significance.
Dfo Reference Standard Usp Deferoxamine Mesylate Reference Standard Lot H, supplied by Pharmacopeia Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/deferoxamine+dfo/dfo+reference+standard+usp+deferoxamine+mesylate+reference+standard+lot+h/10__1128_slash_aac__39__9__2023-27-1-13
Average 90 stars, based on 1 article reviews
dfo reference standard usp deferoxamine mesylate reference standard lot h - by Bioz Stars, 2026-10
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deferoxamine mesylate (dfo)  (AbMole Bioscience)
90
AbMole Bioscience deferoxamine mesylate (dfo)
Curcumin inhibits FTC tumorigenesis. (a) Nthy-ori-3-1 and FTC cells were treated with the indicated concentrations of curcumin for 24 h, and curcumin inhibited cell viability. Cell viability was measured using CCK-8 assays. (b–d) Curcumin suppressed the growth of FTC cells. FTC cells were treated with 0, 10, or 20 μ M curcumin and subjected to colony formation, cell migration, and invasion assays. Scale bars: 100 μ m. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the 0 μ M curcumin group. (e, f) FTC cells were either preincubated with various inhibitors, namely, ferrostatin-1 (Fer-1, 10 μ M), <t>deferoxamine</t> (DFO, 10 μ M), chloroquine (CQ, 5 μ M), autophagy inhibitor (3MA, 1 mM), and Z-VAD-FMK (5 μ M), for 2 h and then treated with 10 or 20 μ M curcumin (Cur) for 24 h or treated with curcumin alone. Cell viability was assessed using CCK-8 assays. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the group receiving curcumin treatment alone. The data are presented as the mean ± standard error of the mean (SEM), n = 3. One-way ANOVA (b–d) and t test (e, f) were used to determine statistical significance.
Deferoxamine Mesylate (Dfo), supplied by AbMole Bioscience, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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deferoxamine mesylate (dfo) - by Bioz Stars, 2026-10
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deferoxamine dfo  (Pharma Science Nutrients Inc)
90
Pharma Science Nutrients Inc deferoxamine dfo
Curcumin inhibits FTC tumorigenesis. (a) Nthy-ori-3-1 and FTC cells were treated with the indicated concentrations of curcumin for 24 h, and curcumin inhibited cell viability. Cell viability was measured using CCK-8 assays. (b–d) Curcumin suppressed the growth of FTC cells. FTC cells were treated with 0, 10, or 20 μ M curcumin and subjected to colony formation, cell migration, and invasion assays. Scale bars: 100 μ m. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the 0 μ M curcumin group. (e, f) FTC cells were either preincubated with various inhibitors, namely, ferrostatin-1 (Fer-1, 10 μ M), <t>deferoxamine</t> (DFO, 10 μ M), chloroquine (CQ, 5 μ M), autophagy inhibitor (3MA, 1 mM), and Z-VAD-FMK (5 μ M), for 2 h and then treated with 10 or 20 μ M curcumin (Cur) for 24 h or treated with curcumin alone. Cell viability was assessed using CCK-8 assays. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the group receiving curcumin treatment alone. The data are presented as the mean ± standard error of the mean (SEM), n = 3. One-way ANOVA (b–d) and t test (e, f) were used to determine statistical significance.
Deferoxamine Dfo, supplied by Pharma Science Nutrients Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/deferoxamine+dfo/deferoxamine+dfo/pm28935635-51-19-23
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deferoxamine dfo - by Bioz Stars, 2026-10
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deferoxamine mesylate (dfo)  (InnoChem Inc)
90
InnoChem Inc deferoxamine mesylate (dfo)
Curcumin inhibits FTC tumorigenesis. (a) Nthy-ori-3-1 and FTC cells were treated with the indicated concentrations of curcumin for 24 h, and curcumin inhibited cell viability. Cell viability was measured using CCK-8 assays. (b–d) Curcumin suppressed the growth of FTC cells. FTC cells were treated with 0, 10, or 20 μ M curcumin and subjected to colony formation, cell migration, and invasion assays. Scale bars: 100 μ m. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the 0 μ M curcumin group. (e, f) FTC cells were either preincubated with various inhibitors, namely, ferrostatin-1 (Fer-1, 10 μ M), <t>deferoxamine</t> (DFO, 10 μ M), chloroquine (CQ, 5 μ M), autophagy inhibitor (3MA, 1 mM), and Z-VAD-FMK (5 μ M), for 2 h and then treated with 10 or 20 μ M curcumin (Cur) for 24 h or treated with curcumin alone. Cell viability was assessed using CCK-8 assays. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the group receiving curcumin treatment alone. The data are presented as the mean ± standard error of the mean (SEM), n = 3. One-way ANOVA (b–d) and t test (e, f) were used to determine statistical significance.
Deferoxamine Mesylate (Dfo), supplied by InnoChem Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Image Search Results


Curcumin inhibits FTC tumorigenesis. (a) Nthy-ori-3-1 and FTC cells were treated with the indicated concentrations of curcumin for 24 h, and curcumin inhibited cell viability. Cell viability was measured using CCK-8 assays. (b–d) Curcumin suppressed the growth of FTC cells. FTC cells were treated with 0, 10, or 20 μ M curcumin and subjected to colony formation, cell migration, and invasion assays. Scale bars: 100 μ m. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the 0 μ M curcumin group. (e, f) FTC cells were either preincubated with various inhibitors, namely, ferrostatin-1 (Fer-1, 10 μ M), deferoxamine (DFO, 10 μ M), chloroquine (CQ, 5 μ M), autophagy inhibitor (3MA, 1 mM), and Z-VAD-FMK (5 μ M), for 2 h and then treated with 10 or 20 μ M curcumin (Cur) for 24 h or treated with curcumin alone. Cell viability was assessed using CCK-8 assays. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the group receiving curcumin treatment alone. The data are presented as the mean ± standard error of the mean (SEM), n = 3. One-way ANOVA (b–d) and t test (e, f) were used to determine statistical significance.

Journal: Oxidative Medicine and Cellular Longevity

Article Title: Curcumin Induces Ferroptosis in Follicular Thyroid Cancer by Upregulating HO-1 Expression

doi: 10.1155/2023/6896790

Figure Lengend Snippet: Curcumin inhibits FTC tumorigenesis. (a) Nthy-ori-3-1 and FTC cells were treated with the indicated concentrations of curcumin for 24 h, and curcumin inhibited cell viability. Cell viability was measured using CCK-8 assays. (b–d) Curcumin suppressed the growth of FTC cells. FTC cells were treated with 0, 10, or 20 μ M curcumin and subjected to colony formation, cell migration, and invasion assays. Scale bars: 100 μ m. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the 0 μ M curcumin group. (e, f) FTC cells were either preincubated with various inhibitors, namely, ferrostatin-1 (Fer-1, 10 μ M), deferoxamine (DFO, 10 μ M), chloroquine (CQ, 5 μ M), autophagy inhibitor (3MA, 1 mM), and Z-VAD-FMK (5 μ M), for 2 h and then treated with 10 or 20 μ M curcumin (Cur) for 24 h or treated with curcumin alone. Cell viability was assessed using CCK-8 assays. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the group receiving curcumin treatment alone. The data are presented as the mean ± standard error of the mean (SEM), n = 3. One-way ANOVA (b–d) and t test (e, f) were used to determine statistical significance.

Article Snippet: Erastin (M2679), ferrostatin-1 (Fer-1, M2698), and deferoxamine (DFO, M5129) were all purchased from AbMole Bioscience Inc. (Texas, USA).

Techniques: CCK-8 Assay, Migration